Post Date December, 03 2023
The prostate is the largest accessory gland in men. It can only play its normal secretory function under the regulation and control of the senior center of the brain to maintain the normal tissue structure of the gland. With the increase of age, the control ability of the nerve center is gradually weakened, the level of male hormone is decreased, and the endocrine imbalance occurs.
Many people are confused about their chronic prostatitis. They do not know how to cause it and whether it can be cured after treatment. He Zhiqiang, director of prostate disease expert at Beijing Shuguang Andrology Hospital, pointed out that although chronic prostatitis is an inflammation, bacterial infection is not the only cause, but only as an external secondary factor. There are also some intrinsic and important pathogenic factors in the prostate itself, which are caused by the functional and organic changes of the gland.
(1) Some people say that the factor of psychological medicine is as high as 50%.
(2) Some patients are transformed from acute prostatitis, but most patients have not gone through the stage of acute infection. The route of bacterial infection is the same as that of acute prostatitis, which is mainly caused by the spread of infection foci in other parts of the body through blood, the direct spread of urinary tract infection and the lymphatic infection. Prostate calculus is often accompanied by chronic inflammatory reaction, but microlithiasis is not easy to be found in X-ray examination, and may be an important source of infection. The main pathogenic bacteria are Escherichia coli, accounting for 80% of chronic bacterial prostatitis, and the others are Klebsiella, Proteus and Pseudomonas. The gram-positive bacteria are mainly enterococcus, and other gram-positive bacteria are still controversial in the etiology of chronic bacterial prostatitis.
(3) Autoimmune factors. Chronic prostatitis has a certain relationship with autoimmune factors, which is because it has been found in patients with arthritis. The presence of anti-prostate antibodies, and at least one positive antigen-antibody system found in serum tests.
(4) Allergic reactions to certain viruses can also lead to inflammation. Autoimmune factors. Chronic prostatitis has a certain relationship with autoimmune factors, which is because it has been found in patients with arthritis. The presence of anti-prostate antibodies, and at least one positive antigen-antibody system found in serum tests.
(5) Endocrine dysfunction. The prostate is the largest accessory gland in men. It can only play its normal secretory function under the regulation and control of the senior center of the brain to maintain the normal tissue structure of the gland. With the increase of age, the control ability of the nerve center is gradually weakened, the level of male hormone is decreased, and the endocrine imbalance occurs. At this time, the gland may undergo degenerative changes, that is, the secretion function of the gland is reduced, and the tissue fibrosis and proliferation, thus providing favorable conditions for bacterial infection.
(6) Disorder of blood circulation of glands. The blood supply of the prostate is very rich. It comes from the upper, middle and lower arteries of the prostate. However, the number of veins that return blood should be less. It needs to pass through many small blood vessels before gradually converging into the large vein vessels, which increases the blood flow resistance in the prostate. At this time, if there are some factors that can make the gland repeatedly congest for a long time, it may aggravate the obstruction of venous reflux. As time goes on, the glands also show delayed changes. Fibrous tissue replaces glandular cells. Gland sclerosis further aggravates the blood circulation disorder in the gland, causing blood stasis and increasing the chance of bacterial retention. Prostate hyperemia due to various reasons, especially passive hyperemia, is an important pathogenic factor. Non-infective and non-microbial hyperemia for a long time can form non-specific inflammatory reaction.
