Postpartum may experience premenstrual syndrome. Grasp 5 points to stay away from the disease

Premenstrual syndrome is a common disease in women, and it often occurs after childbirth. It is necessary to clarify the factors causing the disease, and to prevent life, diet, and mood in a reasonable manner after childbirth.

Causes of postpartum syndrome

1. Fluid accumulation: Excessive aldosterone hormone causes systemic fluid accumulation, commonly used to explain the formation of early syndrome. Progesterone can prevent the effect of aldosterone on the renal tubules, which is beneficial for the excretion of urine sodium. However, due to the loss of sodium caused by progesterone, the compensatory effect of aldosterone increases. In addition, the activity of converting progesterone into haloperidins such as deoxygenate increases in the second half of menstruation, and the secretion of aldosterone increases before menstruation, reaching a peak before menstruation. Therefore, the increase in aldosterone excretion during pregnancy is physiological

2. Psychological stimulation: Due to the widespread and disconnected nature of symptoms in the early stages of the syndrome, using a placebo or receiving psychological and psychological treatment can also be effective. Many scholars have proposed that the reason for mental and social factors causing physical and mental dysfunction is Parker's synthesis of many scholars' opinions. Personality and environmental factors are extremely important for the occurrence of early syndrome symptoms, and the appearance of symptoms reflects unresolved conflicts in the patient's heart. Tracing the patient's life history often reveals significant psychological stimuli, such as childhood misfortunes and trauma, parental and family discord, poor academic performance, and breakdowns, which may be important factors in emotional changes before the event.

3. Prolactin: The increase in prolactin (PRL) emissions: In recent years, the issue of PRL emissions being an important factor in PMS has sparked significant debate. The plasma PRL concentration has a circadian rhythm, with the highest level during sleep and significant fluctuations between days per person. The PRL level reaches its peak during ovulation, and the average PRL level during luteal phase is higher than that during follicular phase. Some early syndrome patients have higher average blood PRL concentrations than healthy women throughout the entire menstrual cycle, especially in the early stages. Treatment with bromocriptine inhibits PRL secretion and significantly alleviates symptoms. The increase in PRL levels during the luteal phase can be accompanied by a decrease in progesterone emissions and a decrease in FSH/LH levels. Theoretically, it is supported that the increase in PRL levels is related to the formation of premenstrual syndrome in some aspects. However, there was no significant difference in PRL levels observed between normal and pre syndrome patients. The regulatory effect of PRL on osmotic pressure is significant in animals and has little impact on humans. It only affects the breast, and the balance of local osmotic pressure may expand the breast and cause pain. In addition, women with high PRL rarely experience symptoms of pre syndrome. The application of bromocriptine therapy only alleviates breast symptoms, while other symptoms have no significant effect. There is a lack of reliable and strong evidence for increasing PRL emissions.