Azoospermia accounts for about 15% - 20% of male infertility patients. One is the dysfunction of the testis itself, which is called primary azoospermia or non obstructive azoospermia. Second, the testicular spermatogenesis function is normal, but the sperm cannot be discharged from the body due to the obstruction of the spermatic duct, which is called obstructive azoospermia.
Azoospermia is the main cause of male infertility, which has a great impact on family harmony. So why does azoospermia occur?
10 factors causing azoospermia
1. Varicocele: Varicocele will lead to high local temperature, microcirculatory disorders, reduced nutrition, decreased partial pressure of oxygen, and lack of nutrient supply, which will affect sexual sperm motility vitality and testicular spermatogenesis.
2. Chromosome abnormality: A gene segment on a chromosome that determines sperm production. If lacking, it can lead to azoospermia and oligospermia.
3. Infections can cause inflammation of the epididymis, vas deferens, seminal vesicles, prostate, and other reproductive ducts, which can affect the motility of sexual sperm and the reproductive function of the testes.
4. Endocrine abnormalities: The pituitary gland of the brain secretes initiators and maintainers for testicular sperm production. If too low, it can lead to decreased testicular spermatogenic function.
5. Genital damage: External injuries such as testicular damage or torsion, spermatic cord torsion, or trauma caused by hernia repair surgery or prostate surgery can also affect testicular function, leading to sperm production disorders. This situation is mainly prevented and effective measures should be taken in a timely manner.
6. Anorchidism: The testicles are the organs that produce sperm in males, and without them, there is no way to talk about sperm.
7. Bilateral cryptorchidism: In this situation, the testicles do not reach the scrotum where they should be, and stay in the abdominal cavity due to the temperature being much higher than the scrotum, making it impossible for sperm to be generated, resulting in azoospermia.
8. Immune factors: Anti sperm antibodies can attach to the tail of sperm, causing a decrease in sperm motility or viability.
9. Environmental factors: In clinical practice, it is common to see a large amount of sperm, long-term exposure to high temperatures, exposure to radiation, and exposure to chemical toxins, which are prone to reproductive dysfunction.
10. Nutritional factors: Amino acids, vitamins A, E, and folic acid deficiency, which are essential nutrients for spermatogenesis, can lead to oligozoospermia and asthenozoospermia.